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Stop blaming your genes for your metabolism. While we’re often told obesity is 70% genetic, pioneering scientists like Professor Denis Noble and Robert Sapolsky prove that DNA is not a master blueprint—it’s a library of "If/Then" switches. Learn why the "Selfish Gene" model is dead and how the "Paris to Leiden" signaling network allows your daily choices to talk back to your genome. From the evolutionary "satiety hack" of Peptide YY (PYY) to the proof found in GLP-1 drugs an

Progesterone and estrogen repeatedly stimulate breast tissue and the uterine lining. Without the “breaks” of pregnancy and lactation, this repeated turnover is thought to increase the risk of fibroids, endometriosis, and possibly breast and endometrial cancers.

Your hormones aren’t the enemy. They’re the original biohack — refined over millions of years to help you survive and thrive. When you learn to work with their rhythm, you unlock a level of ease and effectiveness that no one-size-fits-all diet can match.

Saturated fat became a convenient scapegoat, while the role of refined carbohydrates, ultra-processed foods, and chronic inflammation in driving heart disease was largely overlooked.

While GLP-1 medications may offer a quick fix for weight loss, they come with potential side effects and long-term concerns

Reality is biochemistry determines behaviour. Overeating is not a failure of willpower; it is the result of a biochemical/hormonal imbalance

There is no universal diet or lifestyle that works for everyone. Instead, solutions should be based on the root causes.

The brain, where disrupted signalling leads to a cascade of events that drive overconsumption and ultimately, system insulin resistance.

Why does a cell become insulin resistant? While IR is often blamed on excess weight or poor lifestyle choices, reality is far more complex!

Insulin Resistance is a system-wide metabolic dysfunction that unfolds in a predictable sequence, much like a chain reaction.