CONNECT | GROW | TRANSFORM

This paradigm shift not only redefines how we view cancer’s origins but also opens new doors for prevention and management strategies.

This metabolic reprogramming is not a random mutation-driven process but a fallback strategy encoded in the evolutionary history of life.

We’ve been conditioned to see food primarily as fuel, the truth is, food plays a far richer role in our bodies than simply providing fuel.

From an evolutionary standpoint, fructose may have offered survival advantages by providing a rapid, concentrated source of energy.

Fructose metabolism differs from glucose metabolism, and is particularly adept at promoting fat storage and disrupts metabolic health.

Chronically elevated insulin levels might be a contributing factor to the RalA-DRP1 pathway that leads to mitochondrial fragmentation.

Life is not just a series of isolated mechanisms but a dynamic, self-organising system.

Let's delve into the fascinating concept of insulin resistance as a 2 sided coin, and how it can manifest differently in various cell types

Elevated insulin levels can rise significantly (up to 20 years!) before glucose levels become abnormal. 

It's crucial to note that insulin resistance isn't a uniform phenomenon. Not all cells are affected equally.