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We’ve been told that heart disease is a simple math problem: "Lower your ApoB at all costs." But the latest science suggests we’re looking at the scoreboard instead of the game. Your LDL particles aren't just numbers; they are structural delivery trucks. The real question isn't how many trucks are on the road, but what they are carrying and how stable they are. In Part 2 of our deep dive, we break down the 5 Pillars of Vascular Resilience

In the latest health podcasts, the message is clear: "Lower your ApoB at all costs and replace SFA with seed oils or face the consequences." But what if the number of trucks on the road isn't the problem? What if the problem is the cargo? In Part 1 of this series, we go "under the hood" of human biochemistry to debunk the mainstream narrative. We explore the Five Phases of the Lipid Loop

Populations adapted by developing energy-conserving physiology — efficient fat storage, early insulin resistance (to prioritise glucose for the brain), and lower lean muscle mass. But when this “thrifty” biology meets modern abundance — high-refined-carb diets, low physical activity — the same traits become maladaptive:→ less muscle mass, less subcutaneous storage, more spillover into visceral depots.

Peptides aren’t the future — they’re the present. But the real biohack? Mastering your body’s own signaling before you ever touch a vial. Peptides aren't magic—they're biology hacks. Stack smart, test blood work, and pair with whole foods/sleep.

At this point, it’s worth asking: if polyunsaturated seed oils and plant sterols disrupt red blood cells, fuel oxidation, promote inflammation, and crystallise in plaques — then why are they still recommended as “cardioprotective”?

The demonization of saturated fat wasn't malice but a cascade: panic-fueled hypothesis, flawed cornerstone study, buried null results, and politicized policy. Vegetable oil industries lobbied (visiting scientists to yank papers), while a small cadre controlled grants and journals, stifling dissent.

Progesterone and estrogen repeatedly stimulate breast tissue and the uterine lining. Without the “breaks” of pregnancy and lactation, this repeated turnover is thought to increase the risk of fibroids, endometriosis, and possibly breast and endometrial cancers.

Your hormones aren’t the enemy. They’re the original biohack — refined over millions of years to help you survive and thrive. When you learn to work with their rhythm, you unlock a level of ease and effectiveness that no one-size-fits-all diet can match.

Explore the physiological link between seed oils, linoleic acid, and oxidized LDL. Learn how high triglycerides and metabolic dysfunction turn LDL into "explosive cargo," leading to arterial inflammation and plaque formation. A deep dive into MPO, LOX, and the real cause of heart disease.

What’s actually most prone to oxidation in LDL is the fatty acid tail of the cholesteryl ester in the “oil barrel” (and the PUFA in the phospholipid shell). Cholesterol itself — the rigid steroid ring — is remarkably stable. Pure cholesterol won’t just oxidise easily in the body.